- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2041-1723
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Persistent repair intermediates induce senescence ; volume:9 ; number:1 ; day:25 ; month:9 ; year:2018 ; pages:1-10 ; date:12.2018
Nature Communications ; 9, Heft 1 (25.9.2018), 1-10, 12.2018
- Classification
-
Biowissenschaften, Biologie
- Creator
-
Feringa, F. M.
- Contributor
-
Raaijmakers, J. A.
Hadders, M. A.
Vaarting, C.
Macurek, L.
Heitink, L.
Krenning, L.
Medema, R. H.
SpringerLink (Online service)
- DOI
-
10.1038/s41467-018-06308-9
- URN
-
urn:nbn:de:101:1-2018111000125584491123
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:51 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Feringa, F. M.
- Raaijmakers, J. A.
- Hadders, M. A.
- Vaarting, C.
- Macurek, L.
- Heitink, L.
- Krenning, L.
- Medema, R. H.
- SpringerLink (Online service)
Other Objects (12)
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Dopants Induce Persistent Room Temperature Phosphorescence in Triarylamine Boronate Esters
Dopants Induce Persistent Room Temperature Phosphorescence in Triarylamine Boronate Esters
SORBS2 and TLR3 induce premature senescence in primary human fibroblasts and keratinocytes
Progressive senescence programs induce intrinsic vulnerability to aging-related female breast cancer
High doses of synthetic antioxidants induce premature senescence in cultivated mesenchymal stem cells
Environmentally persistent free radicals induce airway hyperresponsiveness in neonatal rat lungs
Senescence, DNA damage and repair in chronic obstructive pulmonary disease (COPD)
Local H2 release remodels senescence microenvironment for improved repair of injured bone
Bleomycin induces senescence and repression of DNA repair via downregulation of Rad51
Organic intermediates, Supplement
Up-regulated succinylation modifications induce a senescence phenotype in microglia by altering mitochondrial energy metabolism
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Dopants Induce Persistent Room Temperature Phosphorescence in Triarylamine Boronate Esters
Dopants Induce Persistent Room Temperature Phosphorescence in Triarylamine Boronate Esters
SORBS2 and TLR3 induce premature senescence in primary human fibroblasts and keratinocytes
Progressive senescence programs induce intrinsic vulnerability to aging-related female breast cancer
High doses of synthetic antioxidants induce premature senescence in cultivated mesenchymal stem cells
Environmentally persistent free radicals induce airway hyperresponsiveness in neonatal rat lungs
Senescence, DNA damage and repair in chronic obstructive pulmonary disease (COPD)
Local H2 release remodels senescence microenvironment for improved repair of injured bone
Bleomycin induces senescence and repression of DNA repair via downregulation of Rad51
Organic intermediates, Supplement
Up-regulated succinylation modifications induce a senescence phenotype in microglia by altering mitochondrial energy metabolism
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Dopants Induce Persistent Room Temperature Phosphorescence in Triarylamine Boronate Esters
Dopants Induce Persistent Room Temperature Phosphorescence in Triarylamine Boronate Esters
SORBS2 and TLR3 induce premature senescence in primary human fibroblasts and keratinocytes
Progressive senescence programs induce intrinsic vulnerability to aging-related female breast cancer
High doses of synthetic antioxidants induce premature senescence in cultivated mesenchymal stem cells
Environmentally persistent free radicals induce airway hyperresponsiveness in neonatal rat lungs
Senescence, DNA damage and repair in chronic obstructive pulmonary disease (COPD)
Local H2 release remodels senescence microenvironment for improved repair of injured bone
Bleomycin induces senescence and repression of DNA repair via downregulation of Rad51
Organic intermediates, Supplement