Hochschulschrift
Interference with stem cell-specific surveillance mechanisms by mutant Lef1 promotes sebaceous tumour development
- Sprache
-
Englisch
- Umfang
-
130 Blätter
- Maße
-
30 cm
- Anmerkungen
-
Illustrationen
Zsfassung in dt. und engl. Sprache
Universität zu Köln, Dissertation, 2016
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Klassifikation
-
Medizin, Gesundheit
- Inhaltsverzeichnis
- Rechteinformation
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Letzte Aktualisierung
-
11.03.2025, 12:06 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
Entstanden
- 2016
Ähnliche Objekte (12)
XIAP promotes melanoma growth by inducing tumour neutrophil infiltration
Identification of regulators of mutant p53 accumulation in lymphoma using RNA interference
Identification of regulators of mutant p53 accumulation in lymphoma using RNA interference
Establishment of a 3D tumour model and targeted therapy of BRAF-mutant colorectal cancer
Targeting p53 protein function in mutant Lef1 tumour cells exploring a 3D in vitro model
The RET receptor tyrosine kinase promotes AML through protection of mutant FLT3 from autophagic degradation
Interference
Interference
Interference
Specific Interference with the Determination of the Tumour-Associated Glycoprotein 72 by Human Anti-Idiotypic Antibodies Formed after Treatment with the Anti-Tumour-Associated Glycoprotein 72 Antibody B72.3
Surveillance
Computerization always promotes centralization even as it promotes decentralization
XIAP promotes melanoma growth by inducing tumour neutrophil infiltration
Identification of regulators of mutant p53 accumulation in lymphoma using RNA interference
Identification of regulators of mutant p53 accumulation in lymphoma using RNA interference
Establishment of a 3D tumour model and targeted therapy of BRAF-mutant colorectal cancer
Targeting p53 protein function in mutant Lef1 tumour cells exploring a 3D in vitro model
The RET receptor tyrosine kinase promotes AML through protection of mutant FLT3 from autophagic degradation
Interference
Interference
Interference
Specific Interference with the Determination of the Tumour-Associated Glycoprotein 72 by Human Anti-Idiotypic Antibodies Formed after Treatment with the Anti-Tumour-Associated Glycoprotein 72 Antibody B72.3
Surveillance
Computerization always promotes centralization even as it promotes decentralization
XIAP promotes melanoma growth by inducing tumour neutrophil infiltration
Identification of regulators of mutant p53 accumulation in lymphoma using RNA interference
Identification of regulators of mutant p53 accumulation in lymphoma using RNA interference
Establishment of a 3D tumour model and targeted therapy of BRAF-mutant colorectal cancer
Targeting p53 protein function in mutant Lef1 tumour cells exploring a 3D in vitro model
The RET receptor tyrosine kinase promotes AML through protection of mutant FLT3 from autophagic degradation
Interference
Interference
Interference
Specific Interference with the Determination of the Tumour-Associated Glycoprotein 72 by Human Anti-Idiotypic Antibodies Formed after Treatment with the Anti-Tumour-Associated Glycoprotein 72 Antibody B72.3
Surveillance