Retraction Note to: A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2051-5960

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Retraction Note to: A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD ; volume:4 ; number:1 ; day:9 ; month:12 ; year:2016 ; pages:1 ; date:12.2016
Acta Neuropathologica Communications ; 4, Heft 1 (9.12.2016), 1, 12.2016

Creator: Hukema, Renate K.

Contributor: Riemslagh, Fréderike W.
Melhem, Shamiram
van der Linde, Herma C.
Severijnen, Lies-Anne W. F. M.
Edbauer, Dieter
Maas, Alex
Charlet-Berguerand, Nicolas
Willemsen, Rob
van Swieten, John C.
SpringerLink (Online service)

DOI: 10.1186/s40478-016-0401-9

URN: urn:nbn:de:1111-20170107752

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:54 AM CEST

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Hukema, Renate K.
Riemslagh, Fréderike W.
Melhem, Shamiram
van der Linde, Herma C.
Severijnen, Lies-Anne W. F. M.
Edbauer, Dieter
Maas, Alex
Charlet-Berguerand, Nicolas
Willemsen, Rob
van Swieten, John C.
SpringerLink (Online service)

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Retraction Note to: A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD

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Other Objects (12)

A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD

A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma

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A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma

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A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD

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Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell

Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD

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