- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1944-446X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Implications of vessel co-option in sorafenib-resistant hepatocellular carcinoma ; volume:35 ; number:1 ; day:25 ; month:11 ; year:2016 ; pages:1-4 ; date:12.2016
Chinese journal of cancer ; 35, Heft 1 (25.11.2016), 1-4, 12.2016
- Creator
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Kuczynski, Elizabeth A.
- Contributor
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Kerbel, Robert S.
SpringerLink (Online service)
- DOI
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10.1186/s40880-016-0162-7
- URN
-
urn:nbn:de:1111-201612151048
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kuczynski, Elizabeth A.
- Kerbel, Robert S.
- SpringerLink (Online service)
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Application of self-assembly peptides targeting the mitochondria as a novel treatment for sorafenib-resistant hepatocellular carcinoma cells
KSR2-14–3-3ζ complex serves as a biomarker and potential therapeutic target in sorafenib-resistant hepatocellular carcinoma
Cytoskeletal gene alterations linked to sorafenib resistance in hepatocellular carcinoma
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CD24 regulates sorafenib resistance via activating autophagy in hepatocellular carcinoma
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Novel FLT3/AURK multikinase inhibitor is efficacious against sorafenib-refractory and sorafenib-resistant hepatocellular carcinoma
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Unresectable Ectopic Hepatocellular Carcinoma Treated with Sorafenib
ID1-induced p16/IL6 axis activation contributes to the resistant of hepatocellular carcinoma cells to sorafenib
Metallothionein-3 is a multifunctional driver that modulates the development of sorafenib-resistant phenotype in hepatocellular carcinoma cells
Application of self-assembly peptides targeting the mitochondria as a novel treatment for sorafenib-resistant hepatocellular carcinoma cells
KSR2-14–3-3ζ complex serves as a biomarker and potential therapeutic target in sorafenib-resistant hepatocellular carcinoma
Cytoskeletal gene alterations linked to sorafenib resistance in hepatocellular carcinoma
Sorafenib-Induced Apoptosis in Hepatocellular Carcinoma Is Reversed by SIRT1
CD24 regulates sorafenib resistance via activating autophagy in hepatocellular carcinoma
Cost-effectiveness of sorafenib versus SBRT for unresectable advanced hepatocellular carcinoma