IL-33, IL-25 and TSLP contribute to development of fungal-associated protease-induced innate-type airway inflammation

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: IL-33, IL-25 and TSLP contribute to development of fungal-associated protease-induced innate-type airway inflammation ; volume:8 ; number:1 ; day:21 ; month:12 ; year:2018 ; pages:1-12 ; date:12.2018
Scientific reports ; 8, Heft 1 (21.12.2018), 1-12, 12.2018

Creator: Hiraishi, Yoshihisa
Yamaguchi, Sachiko
Yoshizaki, Takamichi
Nambu, Aya
Shimura, Eri
Takamori, Ayako
Narushima, Seiko
Nakanishi, Wakako
Asada, Yosuke
Numata, Takafumi
Suzukawa, Maho
Yamauchi, Yasuhiro
Matsuda, Akira
Arae, Ken
Morita, Hideaki
Hoshino, Tomoaki
Suto, Hajime
Okumura, Ko
Matsumoto, Kenji
Saito, Hirohisa
Sudo, Katsuko
Iikura, Motoyasu
Nagase, Takahide
Nakae, Susumu

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41598-018-36440-x

URN: urn:nbn:de:101:1-2019020602554266952312

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:50 AM CEST

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Associated

Hiraishi, Yoshihisa
Yamaguchi, Sachiko
Yoshizaki, Takamichi
Nambu, Aya
Shimura, Eri
Takamori, Ayako
Narushima, Seiko
Nakanishi, Wakako
Asada, Yosuke
Numata, Takafumi
Suzukawa, Maho
Yamauchi, Yasuhiro
Matsuda, Akira
Arae, Ken
Morita, Hideaki
Hoshino, Tomoaki
Suto, Hajime
Okumura, Ko
Matsumoto, Kenji
Saito, Hirohisa
Sudo, Katsuko
Iikura, Motoyasu
Nagase, Takahide
Nakae, Susumu
SpringerLink (Online service)

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IL-33, IL-25 and TSLP contribute to development of fungal-associated protease-induced innate-type airway inflammation

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Other Objects (12)

Ageing and smoking contribute to plasma surfactant proteins and protease imbalance with correlations to airway obstruction

IL-36γ/IL-1F9, an innate T-bet target in myeloid cells

The IL-10 polarized cytokine pattern in innate and adaptive immunity cells contribute to the development of FVIII inhibitors

IL-13 impairs tight junctions in airway epithelia

Innate Immune Cell Functions Contribute to Spontaneous HIV Control

IFN-γ, IL-4 and IL-13 modulate responsiveness of human airway smooth muscle cells to IL-13

Airway IL-1β associates with IL-5 production following dust mite allergen inhalation in humans

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

IL-6R shedding and its role in innate and adaptive immunity

Herstellung eines IL-6R und TNF spezifischen Inhibitors der ADAM17 Protease

Ageing and smoking contribute to plasma surfactant proteins and protease imbalance with correlations to airway obstruction

IL-36γ/IL-1F9, an innate T-bet target in myeloid cells

The IL-10 polarized cytokine pattern in innate and adaptive immunity cells contribute to the development of FVIII inhibitors

IL-13 impairs tight junctions in airway epithelia

Innate Immune Cell Functions Contribute to Spontaneous HIV Control

IFN-γ, IL-4 and IL-13 modulate responsiveness of human airway smooth muscle cells to IL-13

Airway IL-1β associates with IL-5 production following dust mite allergen inhalation in humans

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

IL-6R shedding and its role in innate and adaptive immunity

Herstellung eines IL-6R und TNF spezifischen Inhibitors der ADAM17 Protease

Ageing and smoking contribute to plasma surfactant proteins and protease imbalance with correlations to airway obstruction

IL-36γ/IL-1F9, an innate T-bet target in myeloid cells

The IL-10 polarized cytokine pattern in innate and adaptive immunity cells contribute to the development of FVIII inhibitors

IL-13 impairs tight junctions in airway epithelia

Innate Immune Cell Functions Contribute to Spontaneous HIV Control

IFN-γ, IL-4 and IL-13 modulate responsiveness of human airway smooth muscle cells to IL-13

Airway IL-1β associates with IL-5 production following dust mite allergen inhalation in humans

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

Interleukin-11 (IL-11) receptor cleavage by the rhomboid protease RHBDL2 induces IL-11 trans-signaling

IL-6R shedding and its role in innate and adaptive immunity

Herstellung eines IL-6R und TNF spezifischen Inhibitors der ADAM17 Protease

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