CD56+ monocytes have a dysregulated cytokine response to lipopolysaccharide and accumulate in rheumatoid arthritis and immunosenescence
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1478-6354
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
CD56+ monocytes have a dysregulated cytokine response to lipopolysaccharide and accumulate in rheumatoid arthritis and immunosenescence ; volume:15 ; number:5 ; day:1 ; month:10 ; year:2013 ; pages:1-8 ; date:10.2013
Arthritis Research & Therapy ; 15, Heft 5 (1.10.2013), 1-8, 10.2013
- Creator
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Krasselt, Marco
- Contributor
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Baerwald, Christoph
Wagner, Ulf
Rossol, Manuela
SpringerLink (Online service)
- DOI
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10.1186/ar4321
- URN
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urn:nbn:de:1111-2016050326619
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:01 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Krasselt, Marco
- Baerwald, Christoph
- Wagner, Ulf
- Rossol, Manuela
- SpringerLink (Online service)
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CD56+ Monocytes Have a Dysregulated Cytokine Response to LPS and Accumulate in Rheumatoid Arthritis and Immunosenescence
Linagliptin inhibits lipopolysaccharide-induced inflammation in human U937 monocytes
Lipopolysaccharide inhalation recruits monocytes and dendritic cell subsets to the alveolar airspace
Systems profiling reveals recurrently dysregulated cytokine signaling responses in ER+ breast cancer patients’ blood
Activation of monocytes and cytokine production in patients with peripheral atherosclerosis obliterans
Epigenetic and transcriptional regulation of cytokine production by Plasmodium falciparum-exposed monocytes
Lipopolysaccharide induces the expression of an autocrine prolactin loop enhancing inflammatory response in monocytes
Is gene expression among women with rheumatoid arthritis dysregulated during a postpartum flare?
Nanoplastics affect the inflammatory cytokine release by primary human monocytes and dendritic cells
Δ9-Tetrahydrocannabinol (THC) enhances lipopolysaccharide-stimulated tissue factor in human monocytes and monocyte-derived microvesicles
LncRNA HOTAIR regulates lipopolysaccharide-induced cytokine expression and inflammatory response in macrophages
Mannan-binding lectin regulates dendritic cell maturation and cytokine production induced by lipopolysaccharide
CD56+ Monocytes Have a Dysregulated Cytokine Response to LPS and Accumulate in Rheumatoid Arthritis and Immunosenescence
Linagliptin inhibits lipopolysaccharide-induced inflammation in human U937 monocytes
Lipopolysaccharide inhalation recruits monocytes and dendritic cell subsets to the alveolar airspace
Systems profiling reveals recurrently dysregulated cytokine signaling responses in ER+ breast cancer patients’ blood
Activation of monocytes and cytokine production in patients with peripheral atherosclerosis obliterans
Epigenetic and transcriptional regulation of cytokine production by Plasmodium falciparum-exposed monocytes
Lipopolysaccharide induces the expression of an autocrine prolactin loop enhancing inflammatory response in monocytes
Is gene expression among women with rheumatoid arthritis dysregulated during a postpartum flare?
Nanoplastics affect the inflammatory cytokine release by primary human monocytes and dendritic cells
Δ9-Tetrahydrocannabinol (THC) enhances lipopolysaccharide-stimulated tissue factor in human monocytes and monocyte-derived microvesicles
LncRNA HOTAIR regulates lipopolysaccharide-induced cytokine expression and inflammatory response in macrophages