Retraction Note: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1528-3658
- Extent
-
Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Retraction Note: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity ; volume:26 ; number:1 ; day:30 ; month:12 ; year:2020 ; pages:1 ; date:12.2020
Molecular medicine ; 26, Heft 1 (30.12.2020), 1, 12.2020
- Creator
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Antoine, Daniel James
Williams, Dominic P.
Kipar, Anja
Laverty, Hugh
Park, B. Kevin
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s10020-020-00262-3
- URN
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urn:nbn:de:101:1-2021020218020463861266
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:31 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Antoine, Daniel James
- Williams, Dominic P.
- Kipar, Anja
- Laverty, Hugh
- Park, B. Kevin
- SpringerLink (Online service)
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Sasa veitchii extracts suppress acetaminophen-induced hepatotoxicity in mice
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The biochemistry of acetaminophen hepatotoxicity and rescue: a mathematical model
High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity
Hepatoprotective effects of safranal on acetaminophen-induced hepatotoxicity in rats
Prolonged treatment with N-acetylcystine delays liver recovery from acetaminophen hepatotoxicity
Ecto-Nucleotide Triphosphate Diphosphohydrolase-2 (NTPDase2) Deletion Increases Acetaminophen-Induced Hepatotoxicity
Recent Updates on Acetaminophen Hepatotoxicity: The Role of Nrf2 in Hepatoprotection
Bifidobacterium adolescentis-derived hypaphorine alleviates acetaminophen hepatotoxicity by promoting hepatic Cry1 expression
A virtual scalable model of the Hepatic Lobule for acetaminophen hepatotoxicity prediction
HMGB1 neutralization is associated with bacterial translocation during acetaminophen hepatotoxicity
Altered responsiveness to extracellular ATP enhances acetaminophen hepatotoxicity
Sasa veitchii extracts suppress acetaminophen-induced hepatotoxicity in mice
Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
The biochemistry of acetaminophen hepatotoxicity and rescue: a mathematical model
High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity
Hepatoprotective effects of safranal on acetaminophen-induced hepatotoxicity in rats
Prolonged treatment with N-acetylcystine delays liver recovery from acetaminophen hepatotoxicity
Ecto-Nucleotide Triphosphate Diphosphohydrolase-2 (NTPDase2) Deletion Increases Acetaminophen-Induced Hepatotoxicity
Recent Updates on Acetaminophen Hepatotoxicity: The Role of Nrf2 in Hepatoprotection
Bifidobacterium adolescentis-derived hypaphorine alleviates acetaminophen hepatotoxicity by promoting hepatic Cry1 expression