METTL3-mediated chromatin contacts promote stress granule phase separation through metabolic reprogramming during senescence
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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METTL3-mediated chromatin contacts promote stress granule phase separation through metabolic reprogramming during senescence ; volume:15 ; number:1 ; day:26 ; month:6 ; year:2024 ; pages:1-16 ; date:12.2024
Nature Communications ; 15, Heft 1 (26.6.2024), 1-16, 12.2024
- Creator
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Wang, Chen
Tanizawa, Hideki
Hill, Connor
Havas, Aaron
Zhang, Qiang
Liao, Liping
Hao, Xue
Lei, Xue
Wang, Lu
Nie, Hao
Qi, Yuan
Tian, Bin
Gardini, Alessandro
Kossenkov, Andrew V.
Goldman, Aaron
Berger, Shelley L.
Noma, Ken-ichi
Adams, Peter D.
Zhang, Rugang
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-024-49745-5
- URN
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urn:nbn:de:101:1-2409162114061.238622639171
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:36 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wang, Chen
- Tanizawa, Hideki
- Hill, Connor
- Havas, Aaron
- Zhang, Qiang
- Liao, Liping
- Hao, Xue
- Lei, Xue
- Wang, Lu
- Nie, Hao
- Qi, Yuan
- Tian, Bin
- Gardini, Alessandro
- Kossenkov, Andrew V.
- Goldman, Aaron
- Berger, Shelley L.
- Noma, Ken-ichi
- Adams, Peter D.
- Zhang, Rugang
- SpringerLink (Online service)
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Reprogramming of mRNA translation in cellular senescence
m6A-methylase METTL3 promotes retinal angiogenesis through modulation of metabolic reprogramming in RPE cells
METTL3-mediated m6A modification of SIRT1 mRNA inhibits progression of endometriosis by cellular senescence enhancing
Senescence-associated reprogramming induced by interleukin-1 impairs response to EGFR neutralization
METTL3 regulates WTAP protein homeostasis
METTL3-mediated m6A modification of SIRT1 mRNA affects the progression of diabetic cataracts through cellular autophagy and senescence
Chemotherapy‐Induced Senescence Reprogramming Promotes Nasopharyngeal Carcinoma Metastasis by circRNA‐Mediated PKR Activation
KMT2A regulates the autophagy-GATA4 axis through METTL3-mediated m6A modification of ATG4a to promote NPCs senescence and IVDD progression
METTL3 promotes osteoblast ribosome biogenesis and alleviates periodontitis
METTL3 Inhibitors for Epitranscriptomic Modulation of Cellular Processes
Pharmacological inhibition of METTL3 impacts specific haematopoietic lineages
Insulin granule biogenesis and exocytosis
Reprogramming of mRNA translation in cellular senescence
m6A-methylase METTL3 promotes retinal angiogenesis through modulation of metabolic reprogramming in RPE cells
METTL3-mediated m6A modification of SIRT1 mRNA inhibits progression of endometriosis by cellular senescence enhancing
Senescence-associated reprogramming induced by interleukin-1 impairs response to EGFR neutralization
METTL3 regulates WTAP protein homeostasis
METTL3-mediated m6A modification of SIRT1 mRNA affects the progression of diabetic cataracts through cellular autophagy and senescence
Chemotherapy‐Induced Senescence Reprogramming Promotes Nasopharyngeal Carcinoma Metastasis by circRNA‐Mediated PKR Activation
KMT2A regulates the autophagy-GATA4 axis through METTL3-mediated m6A modification of ATG4a to promote NPCs senescence and IVDD progression
METTL3 promotes osteoblast ribosome biogenesis and alleviates periodontitis
METTL3 Inhibitors for Epitranscriptomic Modulation of Cellular Processes
Pharmacological inhibition of METTL3 impacts specific haematopoietic lineages