The identification of BCL-XL and MCL-1 as key anti-apoptotic proteins in medulloblastoma that mediate distinct roles in chemotherapy resistance
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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The identification of BCL-XL and MCL-1 as key anti-apoptotic proteins in medulloblastoma that mediate distinct roles in chemotherapy resistance ; volume:14 ; number:10 ; day:28 ; month:10 ; year:2023 ; pages:1-15 ; date:10.2023
Cell death & disease ; 14, Heft 10 (28.10.2023), 1-15, 10.2023
- Classification
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Medizin, Gesundheit
- Creator
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Fitzgerald, Marie-Claire
O’Halloran, Philip J.
Kerrane, Sean A.
Ní Chonghaile, Triona
Connolly, Niamh M. C.
Murphy, Brona M.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41419-023-06231-y
- URN
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urn:nbn:de:101:1-2024011608231702192910
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:38 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Fitzgerald, Marie-Claire
- O’Halloran, Philip J.
- Kerrane, Sean A.
- Ní Chonghaile, Triona
- Connolly, Niamh M. C.
- Murphy, Brona M.
- SpringerLink (Online service)
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Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
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Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL
MCL-1, BCL-xL and Casp-3 as key regulators in thymus homeostasis and thymic epithelial tumors
Regulation of Bcl-xL activity in apoptosis
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
The role of anti-apoptotic BCL-XL protein in the maintenance of human hematopoiesis
Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL
MCL-1, BCL-xL and Casp-3 as key regulators in thymus homeostasis and thymic epithelial tumors
Regulation of Bcl-xL activity in apoptosis
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
The role of anti-apoptotic BCL-XL protein in the maintenance of human hematopoiesis
Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL
MCL-1, BCL-xL and Casp-3 as key regulators in thymus homeostasis and thymic epithelial tumors