Identification of ten variants associated with risk of estrogen-receptor-negative breast cancer
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
In: Nature Genetics, 49, 12, S. 1767-1778
- Event
-
Veröffentlichung
- (where)
-
Augsburg
- (who)
-
Universität Augsburg
- (when)
-
2017
- Event
-
Veröffentlichung
- (who)
-
Springer Science and Business Media LLC
- (when)
-
2017
- Creator
- DOI
-
10.1038/ng.3785
- URN
-
urn:nbn:de:bvb:384-opus4-925497
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Milne, Roger L.
- Landsman, Keren
- Universität Augsburg
- Springer Science and Business Media LLC
Time of origin
- 2017
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Estrogen rescues heart failure through estrogen receptor Beta activation
Regulation of estrogen responsive genes by human estrogen receptor alpha (ERα) [(ER-Alpha)]
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Conservation of estrogen receptor function in invertebrate reproduction
Non-coding single nucleotide variants affecting estrogen receptor binding and activity
Postmenopausal estrogen receptor positive breast cancer and obesity associated gene variants
The therapeutic target of estrogen receptor-alpha36 in estrogen-dependent tumors
Role of Estrogen Receptor-β in Endometriosis
Synthesis of an Estrogen Receptor Beta Agonist
Co-expression of estrogen receptor-alpha and targets of estrogen receptor action in proliferating monkey mammary epithelial cells
An amphioxus orthologue of the estrogen receptor that does not bind estradiol: Insights into estrogen receptor evolution
Estrogen rescues heart failure through estrogen receptor Beta activation
Regulation of estrogen responsive genes by human estrogen receptor alpha (ERα) [(ER-Alpha)]
Breast-related effects of selective estrogen receptor modulators and tissue-selective estrogen complexes
Estrogen-mediated inactivation of FOXO3a by the G protein-coupled estrogen receptor GPER