Correction to: Episodic Ataxia Type 1: Natural History and Effect on Quality of Life
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1473-4230
- Extent
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Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Correction to: Episodic Ataxia Type 1: Natural History and Effect on Quality of Life ; day:29 ; month:7 ; year:2022 ; pages:1
The cerebellum ; (29.7.2022), 1
- Creator
-
Graves, Tracey D.
Griggs, Robert C.
Bundy, Brian N.
Jen, Joanna C.
Baloh, Robert W.
Hanna, Michael G.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s12311-022-01450-z
- URN
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urn:nbn:de:101:1-2022101513252141275566
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:25 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Graves, Tracey D.
- Griggs, Robert C.
- Bundy, Brian N.
- Jen, Joanna C.
- Baloh, Robert W.
- Hanna, Michael G.
- SpringerLink (Online service)
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Episodic Ataxia Type 1: Natural History and Effect on Quality of Life
Episodic Vestibulocerebellar Ataxia Associated with a CACNA1G Missense Variant
A Chinese boy with episodic ataxia and low CSF glucose
Episodic ataxia and severe infantile phenotype in spinocerebellar ataxia type 14: expansion of the phenotype and novel mutations
Episodic ataxia type 2 with a novel missense variant (Leu602Arg) in CACNA1A
A point mutation associated with episodic ataxia 6 increases glutamate transporter anion currents
Correction to: Growth in ataxia telangiectasia
Pathophysiological changes in the hippocampus in a murine model of episodic ataxia type 6
Pin1 promotes human CaV2.1 channel polyubiquitination by RNF138: pathophysiological implication for episodic ataxia type 2
Two distinct phenotypes, hemiplegic migraine and episodic Ataxia type 2, caused by a novel common CACNA1A variant
Apo state pore opening as functional basis of increased EAAT anion channel activity in episodic ataxia 6
A 3-year cohort study of the natural history of spinocerebellar ataxia type 6 in Japan
Episodic Ataxia Type 1: Natural History and Effect on Quality of Life
Episodic Vestibulocerebellar Ataxia Associated with a CACNA1G Missense Variant
A Chinese boy with episodic ataxia and low CSF glucose
Episodic ataxia and severe infantile phenotype in spinocerebellar ataxia type 14: expansion of the phenotype and novel mutations
Episodic ataxia type 2 with a novel missense variant (Leu602Arg) in CACNA1A
A point mutation associated with episodic ataxia 6 increases glutamate transporter anion currents
Correction to: Growth in ataxia telangiectasia
Pathophysiological changes in the hippocampus in a murine model of episodic ataxia type 6
Pin1 promotes human CaV2.1 channel polyubiquitination by RNF138: pathophysiological implication for episodic ataxia type 2
Two distinct phenotypes, hemiplegic migraine and episodic Ataxia type 2, caused by a novel common CACNA1A variant
Apo state pore opening as functional basis of increased EAAT anion channel activity in episodic ataxia 6