Die Interaktion zwischen OMI/HtrA2 und NG2 in oligodendroglialen Vorläuferzellen : NG2 und Homöostase?
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Deutsch
- Notes
-
Mainz, Johannes Gutenberg-Universität, Dissertation, 2012
- Classification
-
Biowissenschaften, Biologie
- Keyword
-
Oxidativer Stress
Gliose
Nervenzelle
Axon
Knockout
Zelle
- Event
-
Veröffentlichung
- (where)
-
Mainz
- (who)
-
Universitätsbibliothek Mainz
- (when)
-
2018
- Creator
- URN
-
urn:nbn:de:hebis:77-diss-1000019458
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:00 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Maus, Frank
- Universitätsbibliothek Mainz
Time of origin
- 2018
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The proteases HtrA2/Omi and UCH-L1 regulate TNF-induced necroptosis
Mutationsanalyse und funktionelle Charakterisierung des Omi-HtrA2-Gens in der Pathogenese der Parkinson-Krankheit
The influence of the PD-associated gene products PINK1 and HtrA2-Omi on mitochondrial integrity
Omi/HtrA2 Regulates a Mitochondria-Dependent Apoptotic Pathway in a Murine Model of Septic Encephalopathy
Expressionsprofil und prognostische Bedeutung der mitochondrialen IAP Antagonisten Smac/DIABLO und Omi/HtrA2 im klarzelligen Nierenzellkarzinom
DUI A2 HTR : Duits A2 voor horeca, toerisme en recreatie
Molecularly imprinted polymers for the analysis of protein phosphorylation and the role of HtrA2/Omi protein in Parkinson's disease
YAP Inhibits the Apoptosis and Migration of Human Rectal Cancer Cells via Suppression of JNK-Drp1-Mitochondrial Fission-HtrA2/Omi Pathways
Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC-1 cell apoptosis via activating the mROS-HtrA2/Omi pathways
Functional characterization of HtrA2 in the pathogenesis of Parkinson's disease
Protease-independent control of parthanatos by HtrA2/Omi
Beteiligung der Serinpeptidase HtrA2/Omi am Zelltodprogramm Parthanatos
The proteases HtrA2/Omi and UCH-L1 regulate TNF-induced necroptosis
Mutationsanalyse und funktionelle Charakterisierung des Omi-HtrA2-Gens in der Pathogenese der Parkinson-Krankheit
The influence of the PD-associated gene products PINK1 and HtrA2-Omi on mitochondrial integrity
Omi/HtrA2 Regulates a Mitochondria-Dependent Apoptotic Pathway in a Murine Model of Septic Encephalopathy
Expressionsprofil und prognostische Bedeutung der mitochondrialen IAP Antagonisten Smac/DIABLO und Omi/HtrA2 im klarzelligen Nierenzellkarzinom
DUI A2 HTR : Duits A2 voor horeca, toerisme en recreatie
Molecularly imprinted polymers for the analysis of protein phosphorylation and the role of HtrA2/Omi protein in Parkinson's disease
YAP Inhibits the Apoptosis and Migration of Human Rectal Cancer Cells via Suppression of JNK-Drp1-Mitochondrial Fission-HtrA2/Omi Pathways
Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC-1 cell apoptosis via activating the mROS-HtrA2/Omi pathways
Functional characterization of HtrA2 in the pathogenesis of Parkinson's disease
Protease-independent control of parthanatos by HtrA2/Omi
Beteiligung der Serinpeptidase HtrA2/Omi am Zelltodprogramm Parthanatos
The proteases HtrA2/Omi and UCH-L1 regulate TNF-induced necroptosis
Mutationsanalyse und funktionelle Charakterisierung des Omi-HtrA2-Gens in der Pathogenese der Parkinson-Krankheit
The influence of the PD-associated gene products PINK1 and HtrA2-Omi on mitochondrial integrity
Omi/HtrA2 Regulates a Mitochondria-Dependent Apoptotic Pathway in a Murine Model of Septic Encephalopathy
Expressionsprofil und prognostische Bedeutung der mitochondrialen IAP Antagonisten Smac/DIABLO und Omi/HtrA2 im klarzelligen Nierenzellkarzinom
DUI A2 HTR : Duits A2 voor horeca, toerisme en recreatie
Molecularly imprinted polymers for the analysis of protein phosphorylation and the role of HtrA2/Omi protein in Parkinson's disease
YAP Inhibits the Apoptosis and Migration of Human Rectal Cancer Cells via Suppression of JNK-Drp1-Mitochondrial Fission-HtrA2/Omi Pathways
Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC-1 cell apoptosis via activating the mROS-HtrA2/Omi pathways