Frequent mutations in acetylation and ubiquitination sites suggest novel driver mechanisms of cancer
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1756-994X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Frequent mutations in acetylation and ubiquitination sites suggest novel driver mechanisms of cancer ; volume:8 ; number:1 ; day:12 ; month:5 ; year:2016 ; pages:1-13 ; date:12.2016
Genome medicine ; 8, Heft 1 (12.5.2016), 1-13, 12.2016
- Creator
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Narayan, Soumil
- Contributor
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Bader, Gary D.
Reimand, Jüri
SpringerLink (Online service)
- DOI
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10.1186/s13073-016-0311-2
- URN
-
urn:nbn:de:1111-2016051718698
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:58 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Narayan, Soumil
- Bader, Gary D.
- Reimand, Jüri
- SpringerLink (Online service)
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KEAP1 Mutations Drive Tumorigenesis by Suppressing SOX9 Ubiquitination and Degradation
Frequent Mutations in Natural Killer/T Cell Lymphoma
Acetylation- and ubiquitination-regulated SFMBT2 acts as a tumor suppressor in clear cell renal cell carcinoma
Exosomal circ_0026611 contributes to lymphangiogenesis by reducing PROX1 acetylation and ubiquitination in human lymphatic endothelial cells (HLECs)
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Different molecular features suggest distinct disease-causing mechanisms in MPL gain-of-function mutations
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Inherited and multiple de novo mutations in autism/developmental delay risk genes suggest a multifactorial model
N-terminal acetylation can stabilize proteins independent of their ubiquitination
Interplay between acetylation and ubiquitination controls PSAT1 protein stability in lung adenocarcinoma
HDAC10 switches NLRP3 modification from acetylation to ubiquitination and attenuates acute inflammatory diseases
KEAP1 Mutations Drive Tumorigenesis by Suppressing SOX9 Ubiquitination and Degradation
Frequent Mutations in Natural Killer/T Cell Lymphoma
Acetylation- and ubiquitination-regulated SFMBT2 acts as a tumor suppressor in clear cell renal cell carcinoma
Exosomal circ_0026611 contributes to lymphangiogenesis by reducing PROX1 acetylation and ubiquitination in human lymphatic endothelial cells (HLECs)
International Reports of Unexpected Low Plasma Concentrations of Dabigatran Suggest That More Frequent Measurements Will Add Value
Different molecular features suggest distinct disease-causing mechanisms in MPL gain-of-function mutations
Erratum: International Reports of Unexpected Low Plasma Concentrations of Dabigatran Suggest That More Frequent Measurements Will Add Value
Suggest Reklame GmbH