A preclinical mice model of multiple sclerosis based on the toxin-induced double-site demyelination of callosal and cerebellar fibers
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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A preclinical mice model of multiple sclerosis based on the toxin-induced double-site demyelination of callosal and cerebellar fibers ; volume:57 ; number:1 ; day:22 ; month:7 ; year:2024 ; pages:1-14 ; date:12.2024
Biological research ; 57, Heft 1 (22.7.2024), 1-14, 12.2024
- Creator
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Vejar, Sebastián
Pizarro, Ignacio S.
Pulgar-Sepúlveda, Raúl
Vicencio, Sinay C.
Polit, Andrés
Amador, Cristian A.
Rio, Rodrigo del
Varas, Rodrigo
Orellana, Juan A.
Ortiz, Fernando C.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s40659-024-00529-7
- URN
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urn:nbn:de:101:1-2410072102002.109249471883
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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13.08.20252025, 8:35 AM CEST
Data provider
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Associated
- Vejar, Sebastián
- Pizarro, Ignacio S.
- Pulgar-Sepúlveda, Raúl
- Vicencio, Sinay C.
- Polit, Andrés
- Amador, Cristian A.
- Rio, Rodrigo del
- Varas, Rodrigo
- Orellana, Juan A.
- Ortiz, Fernando C.
- SpringerLink (Online service)
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Astroglial and therapeutic factors affect demyelination in murine models with toxic demyelination
Autotaxin and lysophosphatidic acid1 receptor-mediated demyelination of dorsal root fibers by sciatic nerve injury and intrathecal lysophosphatidylcholine
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Abnormal eyeblink conditioning is an early marker of cerebellar dysfunction in preclinical SCA3 mutation carriers
The chemokine fractalkine (CX3CL1) attenuates H2O2-induced demyelination in cerebellar slices
Tenascins interfere with remyelination in an ex vivo cerebellar explant model of demyelination
Experimental neurocysticercosis and demyelination
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Astroglial and therapeutic factors affect demyelination in murine models with toxic demyelination
Autotaxin and lysophosphatidic acid1 receptor-mediated demyelination of dorsal root fibers by sciatic nerve injury and intrathecal lysophosphatidylcholine
Neuronal hibernation following hippocampal demyelination
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